STK38L kinase ablation promotes loss of cell viability in a subset of KRAS-dependent pancreatic cancer Cell lines
Pancreatic ductal adenocarcinomas (PDACs) are highly aggressive malignancies, associated with poor clinical prognosis and limited therapeutic options. The KRAS oncogene is mutated in over 90% of PDACs and plays a pivotal role in tumor progression. Global gene expression profiling of PDAC reveals 3-4...
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| Language: | en_US |
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2018
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| Online Access: | https://hdl.handle.net/2144/26511 |