Targeting adenosine monophosphate-activated protein kinase (AMPK) in preclinical models reveals a potential mechanism for the treatment of neuropathic pain

Targeting adenosine monophosphate-activated protein kinase (AMPK) in preclinical models reveals a potential mechanism for the treatment of neuropathic pain

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Neuropathic pain is a debilitating clinical condition with few efficacious treatments, warranting development of novel therapeutics. We hypothesized that dysregulated translation regulation pathways may underlie neuropathic pain. Peripheral nerve injury induced reorganization of translation machiner...

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Bibliographic Details
Main Authors: Melemedjian, Ohannes, Asiedu, Marina, Tillu, Dipti, Sanoja, Raul, Yan, Jin, Lark, Arianna, Khoutorsky, Arkady, Johnson, Jessica, Peebles, Katherine, Lepow, Talya, Sonenberg, Nahum, Dussor, Gregory, Price, Theodore
Other Authors: Department of Pharmacology, University of Arizona, N Campbell Ave, Tucson, 85724, USA
Language:en
Published: BioMed Central 2011
Online Access:Melemedjian et al. Molecular Pain 2011, 7:70 http://www.molecularpain.com/content/7/1/70
http://hdl.handle.net/10150/610214
http://arizona.openrepository.com/arizona/handle/10150/610214

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Melemedjian et al. Molecular Pain 2011, 7:70 http://www.molecularpain.com/content/7/1/70
http://hdl.handle.net/10150/610214
http://arizona.openrepository.com/arizona/handle/10150/610214

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