The effect of pharmacological inhibition of mitogen- and stress-activated protein kinase-1 (MSK1) on chemokine-induced neutrophil recruitment
Neutrophil recruitment to the site of acute inflammation is a multistep process regulated by specific signaling molecules. The signaling mechanisms that regulate neutrophil-endothelial cell interactions remain incompletely understood. p38 mitogen-activated protein kinase (MAPK) signalling was shown...
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Language: | English |
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2015
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Online Access: | http://hdl.handle.net/10388/ETD-2014-09-1675 |