Nitric oxide-induced cardiomyocyte cell death
Nitric oxide (NO), a regulator of diverse cardiovascular functions, modifies cardiac cell viability through mechanisms that remain uncertain. Several pathways were studied to understand these effects. The possibility that the protein p53 is involved in the cardiomyocyte response to the NO donor s...
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Language: | English |
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University of British Columbia
2011
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Online Access: | http://hdl.handle.net/2429/31539 |