Proton-sensing GPCR-mediated calcium signals regulate the transition from acute to chronic pain.
博士 === 國立中央大學 === 生命科學系 === 104 === Tissue injury and inflammation raise local proton concentration (called tissue acidosis) and accompany with painful sensations. Tissue acidosis is a dominant factor that contributes to pain. Transient receptor potential vanilloid 1 (TRPV1) and acid-sensing ion cha...
| Main Authors: | , |
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| Other Authors: | |
| Format: | Others |
| Language: | en_US |
| Published: |
2016
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| Online Access: | http://ndltd.ncl.edu.tw/handle/14867259630340321116 |