The adipokine Retnla deficiency increases responsiveness to cardiac repair through adiponectin-rich bone marrow cells

The adipokine Retnla deficiency increases responsiveness to cardiac repair through adiponectin-rich bone marrow cells

Abstract Resistin-like alpha (Retnla) is a member of the resistin family and known to modulate fibrosis and inflammation. Here, we investigated the role of Retnla in the cardiac injury model. Myocardial infarction (MI) was induced in wild type (WT), Retnla knockout (KO), and Retnla transgenic (TG) m...

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Bibliographic Details
Main Authors: Yong Sook Kim, Hyang Hee Cho, Dong Im Cho, Hye-yun Jeong, Soo yeon Lim, Ju Hee Jun, Mi Ra Kim, Bo Gyeong Kang, Meeyoung Cho, Hye-jin Kang, Wan Seok Kang, Goo Taeg Oh, Youngkeun Ahn
Format: Article
Language:English
Published: Nature Publishing Group 2021-03-01
Series:Cell Death and Disease
Online Access:https://doi.org/10.1038/s41419-021-03593-z

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https://doi.org/10.1038/s41419-021-03593-z

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