Long noncoding RNA MALAT1 in exosomes drives regenerative function and modulates inflammation-linked networks following traumatic brain injury

Long noncoding RNA MALAT1 in exosomes drives regenerative function and modulates inflammation-linked networks following traumatic brain injury

Abstract Background Neuroinflammation is a common therapeutic target for traumatic brain injury (TBI) due to its contribution to delayed secondary cell death and has the potential to occur for years after the initial insult. Exosomes from adipose-derived stem cells (hASCs) containing the long noncod...

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Bibliographic Details
Main Authors: Niketa A. Patel, Lauren Daly Moss, Jea-Young Lee, Naoki Tajiri, Sandra Acosta, Charles Hudson, Sajan Parag, Denise R. Cooper, Cesario V. Borlongan, Paula C. Bickford
Format: Article
Language:English
Published: BMC 2018-07-01
Series:Journal of Neuroinflammation
Online Access:http://link.springer.com/article/10.1186/s12974-018-1240-3

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http://link.springer.com/article/10.1186/s12974-018-1240-3

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