A novel mechanism of inhibiting in-stent restenosis with arsenic trioxide drug-eluting stent: Enhancing contractile phenotype of vascular smooth muscle cells via YAP pathway

Objective: Arsenic trioxide (ATO or As2O3) has beneficial effects on suppressing neointimal hyperplasia and restenosis, but the mechanism is still unclear. The goal of this study is to further understand the mechanism of ATO's inhibitory effect on vascular smooth muscle cells (VSMCs). Methods a...

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Bibliographic Details
Main Authors:	Yinping Zhao, Guangchao Zang, Tieying Yin, Xiaoyi Ma, Lifeng Zhou, Lingjuan Wu, Richard Daniel, Yunbing Wang, Juhui Qiu, Guixue Wang
Format:	Article
Language:	English
Published:	KeAi Communications Co., Ltd. 2021-02-01
Series:	Bioactive Materials
Subjects:	Arsenic trioxide (ATO) Bioactive Yes-associated protein (YAP) In-stent restenosis (ISR)
Online Access:	http://www.sciencedirect.com/science/article/pii/S2452199X20301912

Internet

http://www.sciencedirect.com/science/article/pii/S2452199X20301912

A novel mechanism of inhibiting in-stent restenosis with arsenic trioxide drug-eluting stent: Enhancing contractile phenotype of vascular smooth muscle cells via YAP pathway

Internet

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