Silencing of A20 Aggravates Neuronal Death and Inflammation After Traumatic Brain Injury: A Potential Trigger of Necroptosis

Silencing of A20 Aggravates Neuronal Death and Inflammation After Traumatic Brain Injury: A Potential Trigger of Necroptosis

Programmed cell death is an important biological process that plays an indispensable role in traumatic brain injury (TBI). Inhibition of necroptosis, a type of programmed cell death, is pivotal in neuroprotection and in preventing associated inflammatory responses. Our results showed that necroptosi...

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Bibliographic Details
Main Authors: Zhongyuan Bao, Liang Fan, Lin Zhao, Xiupeng Xu, Yinlong Liu, Honglu Chao, Ning Liu, Yongping You, Yan Liu, Xiaoming Wang, Jing Ji
Format: Article
Language:English
Published: Frontiers Media S.A. 2019-09-01
Series:Frontiers in Molecular Neuroscience
Subjects:
necroptosis
inflammation
traumatic brain injury
A20
neuronal death
Online Access:https://www.frontiersin.org/article/10.3389/fnmol.2019.00222/full

Internet

https://www.frontiersin.org/article/10.3389/fnmol.2019.00222/full

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