Maternal selenium deficiency in mice promotes sex‐specific changes to urine flow and renal expression of mitochondrial proteins in adult offspring

Abstract Selenium deficiency during pregnancy can impair fetal development and predispose offspring to thyroid dysfunction. Given that key selenoproteins are highly expressed in the kidney and that poor thyroid health can lead to kidney disease, it is likely that kidney function may be impaired in o...

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Bibliographic Details
Main Authors: Elliott S. Neal, Pierre Hofstee, Montana R. Askew, Nykola L. Kent, Lucy A. Bartho, Anthony V. Perkins, James S. M. Cuffe
Format: Article
Language:English
Published: Wiley 2021-03-01
Series:Physiological Reports
Subjects:
Online Access:https://doi.org/10.14814/phy2.14785