Overexpression of <i>NRF1-742</i> or <i>NRF1-772</i> Reduces Arsenic-Induced Cytotoxicity and Apoptosis in Human HaCaT Keratinocytes

Overexpression of <i>NRF1-742</i> or <i>NRF1-772</i> Reduces Arsenic-Induced Cytotoxicity and Apoptosis in Human HaCaT Keratinocytes

Increasing evidence indicates that human exposure to inorganic arsenic causes cutaneous diseases and skin cancers. Nuclear factor erythroid 2-like 1 (NRF1) belongs to the cap &#8220;n&#8221; collar (CNC) basic-region leucine zipper (bZIP) transcription factor family and regulates antioxidant...

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Bibliographic Details
Main Authors: Shuai Wang, Hao Cheng, Linlin Wang, Rui Zhao, Dawei Guan
Format: Article
Language:English
Published: MDPI AG 2020-03-01
Series:International Journal of Molecular Sciences
Subjects:
nrf1
arsenic
hacat cells
cytotoxicity
post-translational modification
Online Access:https://www.mdpi.com/1422-0067/21/6/2014

Internet

https://www.mdpi.com/1422-0067/21/6/2014

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