MKP-3 suppresses LPS-induced inflammatory responses in HUVECs via inhibition of p38 MAPK/NF-κB pathway

MKP-3 suppresses LPS-induced inflammatory responses in HUVECs via inhibition of p38 MAPK/NF-κB pathway

Endothelial cell dysfunction and inflammatory responses play critical roles in the development of atherosclerosis. Recent data on the processes underlying atherogenesis indicate the substantial role of endotoxins (lipopolysaccharides; LPS) of the intestinal microflora in the initiation and progressi...

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Bibliographic Details
Main Authors: Banzragchgarav Unenkhuu, Da Bin Kim, Hong Seok Kim
Format: Article
Language:English
Published: Taylor & Francis Group 2021-07-01
Series:Animal Cells and Systems
Subjects:
mkp-3
endothelial cell
inflammation
lipopolysaccharide
endotoxemia
Online Access:http://dx.doi.org/10.1080/19768354.2021.1954551

Internet

http://dx.doi.org/10.1080/19768354.2021.1954551

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