N-n-Butyl Haloperidol Iodide Ameliorates Cardiomyocytes Hypoxia/Reoxygenation Injury by Extracellular Calcium-Dependent and -Independent Mechanisms

N-n-Butyl Haloperidol Iodide Ameliorates Cardiomyocytes Hypoxia/Reoxygenation Injury by Extracellular Calcium-Dependent and -Independent Mechanisms

N-n-butyl haloperidol iodide (F2) has been shown to antagonize myocardial ischemia/reperfusion injury by blocking calcium channels. This study explores the biological functions of ERK pathway in cardiomyocytes hypoxia/reoxygenation injury and clarifies the mechanisms by which F2 ameliorates cardiomy...

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Bibliographic Details
Main Authors: Yanmei Zhang, Gaoyong Chen, Shuping Zhong, Fuchun Zheng, Fenfei Gao, Yicun Chen, Zhanqin Huang, Wenfeng Cai, Weiqiu Li, Xingping Liu, Yanshan Zheng, Han Xu, Ganggang Shi
Format: Article
Language:English
Published: Hindawi Limited 2013-01-01
Series:Oxidative Medicine and Cellular Longevity
Online Access:http://dx.doi.org/10.1155/2013/912310

Internet

http://dx.doi.org/10.1155/2013/912310

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