Recent treatment of postischaemic anoxic brain damage after cardiac arrest by using therapeutic hypothermia
Organ injury caused by ischemia and anoxia during prolonged cardiac arrest is compounded by reperfusion injury that occurs when spontaneous circulation is restored. Mild hypothermia (32-35ºC) is neuroprotective through several mechanisms, including suppression of apoptosis, reduced production of exc...
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| Format: | Article |
| Language: | English |
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Serbian Medical Society
2008-01-01
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| Series: | Srpski Arhiv za Celokupno Lekarstvo |
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| Online Access: | http://www.doiserbia.nb.rs/img/doi/0370-8179/2008/0370-81790810549A.pdf |