Phospholipid derived mediators and glomerulonephritis

Phospholipid derived mediators and glomerulonephritis

The contributions made by the various eicosanoids, PAF, the HETES and the lipoxins to the pathophysiology of glomerulonephritis is reviewed. A case can be made for clinical trials of PAF, leukotriene and thromboxane antagonists. Combined thromboxane synthetase and thromboxane receptor antagonism wou...

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Main Author: E. N. Wardle
Format: Article
Language:English
Published: Hindawi Limited 1993-01-01
Series:Mediators of Inflammation
Online Access:http://dx.doi.org/10.1155/S0962935193000134
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spelling doaj-5e890d07dcdb4669934e5d3992f6439e2020-11-24T23:10:31ZengHindawi LimitedMediators of Inflammation0962-93511466-18611993-01-01229910210.1155/S0962935193000134Phospholipid derived mediators and glomerulonephritisE. N. Wardle021 Common Road, North Leigh, Oxon, OX8 6RD, UKThe contributions made by the various eicosanoids, PAF, the HETES and the lipoxins to the pathophysiology of glomerulonephritis is reviewed. A case can be made for clinical trials of PAF, leukotriene and thromboxane antagonists. Combined thromboxane synthetase and thromboxane receptor antagonism would seem to be the more efficacious approach for the various disease entities.http://dx.doi.org/10.1155/S0962935193000134
collection DOAJ
language English
format Article
sources DOAJ
author E. N. Wardle
spellingShingle E. N. Wardle
Phospholipid derived mediators and glomerulonephritis
Mediators of Inflammation
author_facet E. N. Wardle
author_sort E. N. Wardle
title Phospholipid derived mediators and glomerulonephritis
title_short Phospholipid derived mediators and glomerulonephritis
title_full Phospholipid derived mediators and glomerulonephritis
title_fullStr Phospholipid derived mediators and glomerulonephritis
title_full_unstemmed Phospholipid derived mediators and glomerulonephritis
title_sort phospholipid derived mediators and glomerulonephritis
publisher Hindawi Limited
series Mediators of Inflammation
issn 0962-9351
1466-1861
publishDate 1993-01-01
description The contributions made by the various eicosanoids, PAF, the HETES and the lipoxins to the pathophysiology of glomerulonephritis is reviewed. A case can be made for clinical trials of PAF, leukotriene and thromboxane antagonists. Combined thromboxane synthetase and thromboxane receptor antagonism would seem to be the more efficacious approach for the various disease entities.
url http://dx.doi.org/10.1155/S0962935193000134
work_keys_str_mv AT enwardle phospholipidderivedmediatorsandglomerulonephritis
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