The modulation of TRPM7 currents by nafamostat mesilate depends directly upon extracellular concentrations of divalent cations
<p>Abstract</p> <p>Concentrations of extracellular divalent cations (Ca<sup>2+ </sup>and Mg<sup>2+</sup>) fall substantially during intensive synaptic transmission as well as during some pathophysiological conditions such as epilepsy and brain ischemia. Here...
Main Authors: | Chen Xuanmao, Numata Tomohiro, Li Minghua, Mori Yasuo, Orser Beverley A, Jackson Michael F, Xiong Zhi-Gang, MacDonald John F |
---|---|
Format: | Article |
Language: | English |
Published: |
BMC
2010-12-01
|
Series: | Molecular Brain |
Online Access: | http://www.molecularbrain.com/content/3/1/38 |
Similar Items
-
Cardiac arrest caused by nafamostat mesilate
by: Hyo Shik Kim, et al.
Published: (2016-09-01) -
Comparison of Ulinastatin, Gabexate Mesilate, and Nafamostat Mesilate in Preservation Solution for Islet Isolation
by: Hirofumi Noguchi M.D., Ph.D., et al.
Published: (2012-03-01) -
Nafamostat Mesilate Is an Extremely Potent Inhibitor of Human Tryptase
by: Shuji Mori, et al.
Published: (2003-01-01) -
Protective Effect of Nafamostat Mesilate on Injury of Porcine Hepatocytes by Human Plasma
by: Yasushi Kawazoe, et al.
Published: (1999-07-01) -
Cardiac arrest caused by nafamostat mesilate: Kounis syndrome in the dialysis room?
by: Ioanna Koniari, et al.
Published: (2017-03-01)