Repeated administration of fenitrothion alters renal functions via oxidative stress mechanism without inhibiting acetylcholinesterase activity in rats
Background: The main mechanism of fenitrothion (FNT) toxicity is by inhibiting the acetylcholinesterase enzyme, though studies have shown that FNT might also develop other toxicological manifestations, including oxidative stress. However, the kidney functions as well as oxidative stress status after...
Main Authors: | , , , |
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Format: | Article |
Language: | English |
Published: |
Biome Scientia
2020-11-01
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Series: | Life Sciences, Medicine and Biomedicine |
Subjects: | |
Online Access: | http://biomescientia.com/index.php/lsmb/article/view/70 |