Kinin-B2 receptor mediated neuroprotection after NMDA excitotoxicity is reversed in the presence of kinin-B1 receptor agonists.

Kinin-B2 receptor mediated neuroprotection after NMDA excitotoxicity is reversed in the presence of kinin-B1 receptor agonists.

Kinins, with bradykinin and des-Arg(9)-bradykinin being the most important ones, are pro-inflammatory peptides released after tissue injury including stroke. Although the actions of bradykinin are in general well characterized; it remains controversial whether the effects of bradykinin are beneficia...

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Bibliographic Details
Main Authors: Antonio H Martins, Janaina M Alves, Dinely Perez, Marimeé Carrasco, Wilmarie Torres-Rivera, Vesna A Eterović, Pedro A Ferchmin, Henning Ulrich
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2012-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3277507?pdf=render

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http://europepmc.org/articles/PMC3277507?pdf=render

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