Amelioration of Mitochondrial Dysfunction-Induced Insulin Resistance in Differentiated 3T3-L1 Adipocytes via Inhibition of NF-κB Pathways

Amelioration of Mitochondrial Dysfunction-Induced Insulin Resistance in Differentiated 3T3-L1 Adipocytes via Inhibition of NF-κB Pathways

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A growing body of evidence suggests that activation of nuclear factor kappa B (NF-κB) signaling pathways is among the inflammatory mechanism involved in the development of insulin resistance and chronic low-grade inflammation in adipose tissues derived from obese animal and human subjects. Neverthel...

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Bibliographic Details
Main Authors: Mohamad Hafizi Abu Bakar, Mohamad Roji Sarmidi, Cheng Kian Kai, Hasniza Zaman Huri, Harisun Yaakob
Format: Article
Language:English
Published: MDPI AG 2014-12-01
Series:International Journal of Molecular Sciences
Subjects:
adipocytes
mitochondrial dysfunction
inflammation
oxidative stress
insulin resistance
celastrol
nuclear factor kappa B (NF-κB)
Online Access:http://www.mdpi.com/1422-0067/15/12/22227

Internet

http://www.mdpi.com/1422-0067/15/12/22227

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