Alzheimer's associated β-amyloid protein inhibits influenza A virus and modulates viral interactions with phagocytes.

Accumulation of β-Amyloid (βA) is a key pathogenetic factor in Alzheimer's disease; however, the normal function of βA is unknown. Recent studies have shown that βA can inhibit growth of bacteria and fungi. In this paper we show that βA also inhibits replication of seasonal and pandemic strains...

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Bibliographic Details
Main Authors: Mitchell R White, Ruth Kandel, Shweta Tripathi, David Condon, Li Qi, Jeffrey Taubenberger, Kevan L Hartshorn
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2014-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC4079246?pdf=render