Circulating NOS3 modulates left ventricular remodeling following reperfused myocardial infarction.

Circulating NOS3 modulates left ventricular remodeling following reperfused myocardial infarction.

PURPOSE:Nitric oxide (NO) is constitutively produced and released from the endothelium and several blood cell types by the isoform 3 of the NO synthase (NOS3). We have shown that NO protects against myocardial ischemia/reperfusion (I/R) injury and that depletion of circulating NOS3 increases within...

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Main Authors: Simone Gorressen, Manuel Stern, Annette M van de Sandt, Miriam M Cortese-Krott, Jan Ohlig, Tienush Rassaf, Axel Gödecke, Jens W Fischer, Gerd Heusch, Marc W Merx, Malte Kelm
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2015-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC4397096?pdf=render

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http://europepmc.org/articles/PMC4397096?pdf=render

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