SARS-CoV-2 Infection Induces Ferroptosis of Sinoatrial Node Pacemaker Cells

SARS-CoV-2 Infection Induces Ferroptosis of Sinoatrial Node Pacemaker Cells

Background: Increasing evidence suggests that cardiac arrhythmias are frequent clinical features of coronavirus disease 2019 (COVID-19). Sinus node damage may lead to bradycardia. However, it is challenging to explore human sinoatrial node (SAN) pathophysiology due to difficulty in isolating and cul...

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Bibliographic Details
Main Authors: Chen, S. (Author), Chen, Z. (Author), Dong, X. (Author), Evans, T. (Author), Gade, A.R (Author), Han, Y. (Author), Higgins, C.A (Author), Ho, D.D (Author), Hurtado, R. (Author), Lacko, L.A (Author), Nilsson-Payant, B.E (Author), Pitt, G.S (Author), Schwartz, R.E (Author), Sisso, W.J (Author), Sun, X. (Author), Tenoever, B.R (Author), Wang, M. (Author), Yang, L. (Author), Zhu, J. (Author)
Format: Article
Language:English
Published: Lippincott Williams and Wilkins 2022
Subjects:
cardiac muscle cell
COVID-19
ferroptosis
Ferroptosis
human
Humans
metabolism
Myocytes, Cardiac
RNA, viral
SARS-CoV-2
sinoatrial node
Sinoatrial Node
sinus node
stem cells
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