Fe65-amyloid precursor protein signalling and Alzheimer's disease
Deposition of Aβ in amyloid plaques and accumulation of hyperphosphorylated tau in neurofibrillary tangles are hallmark pathologies of Alzheimer’s disease. Changes in APP processing alter Aβ generation and are likely to affect APP function, which may also contribute to Alzheimer’s disease. APP binds...
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King's College London (University of London)
2017
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| Online Access: | http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.702533 |