Fis1 and Bap31 bridge the mitochondria/ER interface to establish a platform for apoptosis induction
Mitochondria and the endoplasmic reticulum (ER) are two organelles that critically contribute to apoptosis induction. It is established that they communicate and several factors for this have been identified. However, how cell death signals are transmitted from mitochondria to ER is unknown. During...
Main Author: | Iwasawa, Ryota |
---|---|
Other Authors: | Grimm, Stefan ; Boobis, Alan |
Published: |
Imperial College London
2011
|
Subjects: | |
Online Access: | http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.534043 |
Similar Items
-
The role of FIS in tyrT transcriptional regulation
by: Lazarus, Linda Ruth
Published: (1992) -
BAP31 Inhibits Cell Adaptation to ER Stress Conditions, Negatively Regulating Autophagy Induction by Interaction with STX17
by: Kayo Machihara, et al.
Published: (2019-10-01) -
Structural and biophysical characterization of the cytoplasmic domains of human BAP29 and BAP31.
by: Esben M Quistgaard, et al.
Published: (2013-01-01) -
The induction of apoptosis by protein kinase inhibitors
by: McKeague, A. L.
Published: (2003) -
CDIP1-BAP31 Complex Transduces Apoptotic Signals from Endoplasmic Reticulum to Mitochondria under Endoplasmic Reticulum Stress
by: Takushi Namba, et al.
Published: (2013-10-01)