Development of inducible transgenic mouse models for melanoma
MC1R pathways) would provide an up-to-date, superior mouse model able to mimic molecular aetiology of human melanoma to investigate the functions and mechanisms of other genes such as MITF, B-Raf, MC1R etc involved in the development of human melanoma.
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ndltd-bl.uk-oai-ethos.bl.uk-4951422015-08-04T03:26:16ZDevelopment of inducible transgenic mouse models for melanomaYao, Denggao2009MC1R pathways) would provide an up-to-date, superior mouse model able to mimic molecular aetiology of human melanoma to investigate the functions and mechanisms of other genes such as MITF, B-Raf, MC1R etc involved in the development of human melanoma.616.99RL DermatologyUniversity of Glasgowhttp://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495142http://theses.gla.ac.uk/546/Electronic Thesis or Dissertation |
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616.99 RL Dermatology |
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616.99 RL Dermatology Yao, Denggao Development of inducible transgenic mouse models for melanoma |
description |
MC1R pathways) would provide an up-to-date, superior mouse model able to mimic molecular aetiology of human melanoma to investigate the functions and mechanisms of other genes such as MITF, B-Raf, MC1R etc involved in the development of human melanoma. |
author |
Yao, Denggao |
author_facet |
Yao, Denggao |
author_sort |
Yao, Denggao |
title |
Development of inducible transgenic mouse models for melanoma |
title_short |
Development of inducible transgenic mouse models for melanoma |
title_full |
Development of inducible transgenic mouse models for melanoma |
title_fullStr |
Development of inducible transgenic mouse models for melanoma |
title_full_unstemmed |
Development of inducible transgenic mouse models for melanoma |
title_sort |
development of inducible transgenic mouse models for melanoma |
publisher |
University of Glasgow |
publishDate |
2009 |
url |
http://ethos.bl.uk/OrderDetails.do?uin=uk.bl.ethos.495142 |
work_keys_str_mv |
AT yaodenggao developmentofinducibletransgenicmousemodelsformelanoma |
_version_ |
1716815155735560192 |