OGR1, G2A, and GPR4 regulate mechanical hyperalgesia induced by inflammation via distinct ways
碩士 === 國立中央大學 === 生命科學系 === 106 === Chronic inflammatory pain results from inflammation that releases the mediators to activate or modulate the nociceptors, leading to pain and hyperalgesia. Tissue acidosis appears to be a dominant factor in inflammatory pain, and the acidosis signals can be sensed...
| Main Authors: | , |
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| Other Authors: | |
| Format: | Others |
| Language: | en_US |
| Published: |
2017
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| Online Access: | http://ndltd.ncl.edu.tw/handle/xntb6h |