Pressure Distribution in an Eye Model of Angle-Closure Glaucoma

碩士 === 淡江大學 === 機械與機電工程學系碩士班 === 97 === Glaucoma is a leading cause of losing eyesight in the world, second only to cataract. In general, if intraocular pressure rose over the bearable limits of optical inner structure that lead to damage of the optic nerve, visual field loss, and ultimately sigh...

Full description

Bibliographic Details
Main Authors: Hsiang-Chieh Chiu, 邱湘傑
Other Authors: 李宗翰
Format: Others
Language:zh-TW
Published: 2009
Online Access:http://ndltd.ncl.edu.tw/handle/82921318496621582966
id ndltd-TW-097TKU05489052
record_format oai_dc
spelling ndltd-TW-097TKU054890522016-05-04T04:16:43Z http://ndltd.ncl.edu.tw/handle/82921318496621582966 Pressure Distribution in an Eye Model of Angle-Closure Glaucoma 隅角閉鎖型青光眼於玻璃體之壓力分析 Hsiang-Chieh Chiu 邱湘傑 碩士 淡江大學 機械與機電工程學系碩士班 97 Glaucoma is a leading cause of losing eyesight in the world, second only to cataract. In general, if intraocular pressure rose over the bearable limits of optical inner structure that lead to damage of the optic nerve, visual field loss, and ultimately sight loss. Intraocular pressure is mainly determined by the coupling of the production of aqueous humor and the drainage of aqueous humor mainly through the trabecular meshwork located in the anterior chamber angle. Any abnormalities of structure leads to unbalanced process and blockage, the intraocular pressure increased. This study proposes a simulation mechanism of angle-closured glaucoma via elevating pressure applanated against vitreous body. To analyze the pressure of optical inner structure and design a model of complete eyeball including aqueous humor and vitreous body. This process is associated with increased intraocular pressure resulting from malfunction of aqueous drainage structures and malformation of vitreous body. Hence it damages the surrounding structures including optic nerves. 李宗翰 2009 學位論文 ; thesis 66 zh-TW
collection NDLTD
language zh-TW
format Others
sources NDLTD
description 碩士 === 淡江大學 === 機械與機電工程學系碩士班 === 97 === Glaucoma is a leading cause of losing eyesight in the world, second only to cataract. In general, if intraocular pressure rose over the bearable limits of optical inner structure that lead to damage of the optic nerve, visual field loss, and ultimately sight loss. Intraocular pressure is mainly determined by the coupling of the production of aqueous humor and the drainage of aqueous humor mainly through the trabecular meshwork located in the anterior chamber angle. Any abnormalities of structure leads to unbalanced process and blockage, the intraocular pressure increased. This study proposes a simulation mechanism of angle-closured glaucoma via elevating pressure applanated against vitreous body. To analyze the pressure of optical inner structure and design a model of complete eyeball including aqueous humor and vitreous body. This process is associated with increased intraocular pressure resulting from malfunction of aqueous drainage structures and malformation of vitreous body. Hence it damages the surrounding structures including optic nerves.
author2 李宗翰
author_facet 李宗翰
Hsiang-Chieh Chiu
邱湘傑
author Hsiang-Chieh Chiu
邱湘傑
spellingShingle Hsiang-Chieh Chiu
邱湘傑
Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
author_sort Hsiang-Chieh Chiu
title Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
title_short Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
title_full Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
title_fullStr Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
title_full_unstemmed Pressure Distribution in an Eye Model of Angle-Closure Glaucoma
title_sort pressure distribution in an eye model of angle-closure glaucoma
publishDate 2009
url http://ndltd.ncl.edu.tw/handle/82921318496621582966
work_keys_str_mv AT hsiangchiehchiu pressuredistributioninaneyemodelofangleclosureglaucoma
AT qiūxiāngjié pressuredistributioninaneyemodelofangleclosureglaucoma
AT hsiangchiehchiu yújiǎobìsuǒxíngqīngguāngyǎnyúbōlítǐzhīyālìfēnxī
AT qiūxiāngjié yújiǎobìsuǒxíngqīngguāngyǎnyúbōlítǐzhīyālìfēnxī
_version_ 1718255932240035840