Simulation of medication effects on variant mutant SCN5A sodium by blocking potassium channels
碩士 === 中華大學 === 資訊工程學系碩士班 === 93 === Two gene-mutant sodium channels, 1795insD and I1768V, were considered to be the possible molecular markers in the initiation of long QT (LQT) syndrome. The 1795insD, which decreases the channel availability and reduces the rate of recovery from inactivation, and...
| Main Authors: | , |
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| Other Authors: | |
| Format: | Others |
| Language: | zh-TW |
| Published: |
2005
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| Online Access: | http://ndltd.ncl.edu.tw/handle/12479793414618870274 |