| Summary: | 碩士 === 國立雲林科技大學 === 環境與安全工程系碩士班 === 91 === Airborne polycyclic aromatic hydrocarbons (PAHs) are mainly caused by imcomplete combustion of smoking. Parts of PAHs are mutagenic and caricinogenic matters. Recent toxicology studies have shown that chew betel nut may produce oxygen free radical in oral, and the BaP compound may become mutagenic by way of cytochrome P450. Many researches about respiratory transfer in humn, but ther only discussed about aerosol particals, nasopharynx, oral and lung precipitation. Fewly studies about organic microparticals (ex: PAHs) is danger for respiratory and oral system. In our experiment, we want to study these PAHs is relativity about oral cencer, We use the respiratory mass transfer coefficient (kc) and the local nondimensionalized transfer coefficient (Sherwood number, Sh) to explain the interation between PAHs and human oral system.
The result of experiment, the PAHs compound’s concentrative of smoking fluent of oral is more than respiration fluent. The absortion of ΣPAHs decreased by dependent on increasing suface velocity of smoking in oral and respiration. The absortion of Ⅲ/Ⅳ PAHs concentration is the highest than other compound. The PAHs deposition in oral somoking is increasing by dependent on surface veloctty, especially the subclass Ⅳ PAHs. It shows the compound of Ⅳ PAHs may be relativity about the variance of oral cells. The respiratory mass transfer coefficient (kc) of smoking in respiration and oral is all increasing by dependent on increasing surface velocity. The local nondimensionalized transfer coefficient, Sherwood number (Sh)is increasing by dependent on increasing surface rate of smoking rate. The increaning values of subclass Ⅲ/Ⅳ PAHs is more obvious than subclass Ⅰ/Ⅱ PAHs. In our study, the subclass Ⅲ/Ⅳ PAHs is easy precipitated in oral. These compounds may increase variance of oral cells.
|
|---|
