| Summary: | 碩士 === 中山醫學院 === 醫學研究所 === 89 === 英文摘要(Abstract):The role of calcified and uncalcified plaque on teeth as a primary etiologic factor in inflammatory periodontal disease has been demonstrated by epidemiologic, experimmental, and clinical research.(1.4.6.25.28-30) The formation of calculus can be divided into three phases: (1) the initial attchment of bacteria to a tooth via salivary pellicle, (2) the growth and orangization of plaque, and (3) the mineralization of plaque. (1.6.11.29.)
Human dental calculus consist of supra- and subgingival deposits (Cacioppi, J.T., and Barboriak, J.J., 1967). Both supragingival and subgingival calculus result from the mineralization of dental plaque. (1-4.12-14.18-20) The calculus is usually covered with metabolically active plaque. Supragingival calculus can be found on a healthy periodontium teeth as well as on periodontal disease teeth. But subgingival calculus is always found on a destructive periodontal disease teeth(Friskopp J., and Hammarstrom L., 1980).
Samples of 46 patients were examined with the scanning electron microscope. They were fixed, dehydrated, dried, and coated with approximate 300 Å thick gold. (4.34.)
In the experimental group, the calculus was needlelike (4.12.26.), cobblestone-like(4.14.16.22.23.26.), and elongated, and had different shapes and sizes of platelet forms and cuboidal crystals shapes. The plaque appears as short-like rods shapes(2.8.26.), long-like rods shapes(2.3.8.9.26.), filaments shapes(2.3.8.26.), spheric shapes(2.3.8.9.15.26.), spiroids shapes(26.) of bacteria, cuticle epithelium cell(2.3.5.8.9.26.) and blood cell. (2. 5.8. 26.)
The results recommend that removal of calculus is more beneficial. The patients with periodontal disease will have less depopsites trouble if they follow a good oral hygiene(1.25-31.) and a regular recall care regimen. Another way to improve plaque control is to use toothbrush and flossing more often. (2.25.28-30.)
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