The Mechanisms of β-amyloid peptide25-35 induced neurotoxicity
碩士 === 國防醫學院 === 生理學研究所 === 85 === β-amyloid peptide (PAP) or its active residue (25-35 fragment) , the major component of the senile plaques in senile dementia, has been reported to be neurotoxic in vitro and in vivo extensively. Although the path-physiological mechanisms underlying βAP25...
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| Format: | Others |
| Language: | zh-TW |
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1997
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| Online Access: | http://ndltd.ncl.edu.tw/handle/12268403920793655145 |