Complement protein C1q modulates macrophage molecular signaling and inflammatory responses during ingestion of atherogenic lipoproteins
<p>Foam cell formation from arterial intima macrophages and defective clearance of apoptotic foam cells drive the progression of the inflammatory disease atherosclerosis. The role of innate immune protein C1q in autoimmune disease and pathogen defense is well characterized, however its role in...
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| Language: | EN |
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California State University, Long Beach
2016
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| Online Access: | http://pqdtopen.proquest.com/#viewpdf?dispub=10131681 |