Transient Overexpression of {alpha}-Ca2+/Calmodulin-Dependent Protein Kinase II in the Nucleus Accumbens Shell Enhances Behavioral Responding to Amphetamine

Transient Overexpression of {alpha}-Ca2+/Calmodulin-Dependent Protein Kinase II in the Nucleus Accumbens Shell Enhances Behavioral Responding to Amphetamine

Ca[superscript 2+]/calmodulin-dependent protein kinase II (CaMKII) is known to contribute to the expression of psychostimulant sensitization by regulating dopamine (DA) overflow from DA neuron terminals in the nucleus accumbens (NAcc). The present experiments explored the contribution of CaMKII in N...

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Bibliographic Details
Main Authors: Loweth, Jessica A. (Author), Singer, Bryan F. (Author), Baker, Lorinda K. (Author), Wilke, Georgia (Author), Inamine, Hidetoshi (Author), Bubula, Nancy (Author), Alexander, John K. (Author), Carlezon, William A., Jr (Author), Vezina, Paul (Author), Neve, Rachael L. (Contributor)
Other Authors: Massachusetts Institute of Technology. Department of Brain and Cognitive Sciences (Contributor)
Format: Article
Language:English
Published: Society for Neuroscience, 2010-06-25T15:51:42Z.
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