Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?

Objectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings...

Full description

Bibliographic Details
Main Authors: Kadriye Erkan Turan, Tulay Kansu
Format: Article
Language:English
Published: Hindawi Limited 2016-01-01
Series:Journal of Ophthalmology
Online Access:http://dx.doi.org/10.1155/2016/2856128
id doaj-eb60635e235b424980e7c72c6d58445d
record_format Article
spelling doaj-eb60635e235b424980e7c72c6d58445d2020-11-24T23:05:21ZengHindawi LimitedJournal of Ophthalmology2090-004X2090-00582016-01-01201610.1155/2016/28561282856128Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?Kadriye Erkan Turan0Tulay Kansu1Department of Ophthalmology, Faculty of Medicine, Hacettepe University, Ankara, TurkeyDepartment of Neurology, Faculty of Medicine, Hacettepe University, Ankara, TurkeyObjectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings of patients presented with AACE of undetermined etiology with a minimum follow-up of 1 year were retrieved from the medical records and the results analyzed. Results. A series of 9 esotropia cases (age range: 20–43 years) was reviewed. All patients had full duction and versions, without an A-pattern or V-pattern. All patients had esotropia for distance and near. Neurological evaluation in all cases was normal. Among patients, 3 were treated with prisms, 4 were treated with strabismus surgery, and 1 was treated with botulinum toxin injections; 1 patient declined treatment. In treated patients posttreatment sensory testing indicated restoration of binocularity that remained stable throughout follow-up of 1–9 years. The patient that declined treatment had binocular function with base-out prisms. Conclusion. Acute onset esotropia may be seen without a neurological pathology in adults. Good motor and sensory outcomes can be achieved in these patients with AACE of undetermined etiology via surgical and nonsurgical methods.http://dx.doi.org/10.1155/2016/2856128
collection DOAJ
language English
format Article
sources DOAJ
author Kadriye Erkan Turan
Tulay Kansu
spellingShingle Kadriye Erkan Turan
Tulay Kansu
Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
Journal of Ophthalmology
author_facet Kadriye Erkan Turan
Tulay Kansu
author_sort Kadriye Erkan Turan
title Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
title_short Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
title_full Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
title_fullStr Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
title_full_unstemmed Acute Acquired Comitant Esotropia in Adults: Is It Neurologic or Not?
title_sort acute acquired comitant esotropia in adults: is it neurologic or not?
publisher Hindawi Limited
series Journal of Ophthalmology
issn 2090-004X
2090-0058
publishDate 2016-01-01
description Objectives. Acute acquired comitant esotropia (AACE) can be a diagnostic challenge for ophthalmologists and neurologists because of its association with neurological pathologies. Our study describes a series of adult patients with AACE of undetermined etiology. Methods. Data on the clinical findings of patients presented with AACE of undetermined etiology with a minimum follow-up of 1 year were retrieved from the medical records and the results analyzed. Results. A series of 9 esotropia cases (age range: 20–43 years) was reviewed. All patients had full duction and versions, without an A-pattern or V-pattern. All patients had esotropia for distance and near. Neurological evaluation in all cases was normal. Among patients, 3 were treated with prisms, 4 were treated with strabismus surgery, and 1 was treated with botulinum toxin injections; 1 patient declined treatment. In treated patients posttreatment sensory testing indicated restoration of binocularity that remained stable throughout follow-up of 1–9 years. The patient that declined treatment had binocular function with base-out prisms. Conclusion. Acute onset esotropia may be seen without a neurological pathology in adults. Good motor and sensory outcomes can be achieved in these patients with AACE of undetermined etiology via surgical and nonsurgical methods.
url http://dx.doi.org/10.1155/2016/2856128
work_keys_str_mv AT kadriyeerkanturan acuteacquiredcomitantesotropiainadultsisitneurologicornot
AT tulaykansu acuteacquiredcomitantesotropiainadultsisitneurologicornot
_version_ 1725626097159110656