Acute β-N-Methylamino-L-alanine Toxicity in a Mouse Model
The cyanobacterial neurotoxin β-N-methylamino-L-alanine (BMAA) is considered to be an “excitotoxin,” and its suggested mechanism of action is killing neurons. Long-term exposure to L-BMAA is believed to lead to neurodegenerative diseases including Parkinson’s and Alzheimer’s diseases and amyotrophic...
Main Authors: | Maitham Ahmed Al-Sammak, Douglas G. Rogers, Kyle D. Hoagland |
---|---|
Format: | Article |
Language: | English |
Published: |
Hindawi Limited
2015-01-01
|
Series: | Journal of Toxicology |
Online Access: | http://dx.doi.org/10.1155/2015/739746 |
Similar Items
-
β-N-Methylamino-L-alanine (BMAA) Toxicity Is Gender and Exposure-Age Dependent in Rats
by: Laura Louise Scott, et al.
Published: (2017-12-01) -
Cyanobacterial Neurotoxin β-N-Methylamino-L-alanine (BMAA) in Shark Fins
by: John Pablo, et al.
Published: (2012-02-01) -
β-N-methylamino-l-alanine enhances neurotoxicity through multiple mechanisms
by: Doug Lobner, et al.
Published: (2007-02-01) -
Biotransfer of β-N-Methylamino-l-alanine (BMAA) in a Eutrophicated Freshwater Lake
by: Sandra Lage, et al.
Published: (2015-03-01) -
The neurotoxin β-N-methylamino-L-alanine (BMAA) : Sources, bioaccumulation and extraction procedures
by: Ferreira Lage, Sandra
Published: (2016)