Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells

Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells

Bibliographic Details
Main Authors: Shijun Wang, Leilei Ma, Ji'e Yang, Zhen Dong, Jian Wu, Xiancheng Lu, Jiahong Wang, Fangjie Dai, Guowei Tu, Lei Xu, Gang Zhao, Feng Zhang, Yunzeng Zou, Junbo Ge
Format: Article
Language:English
Published: Wiley 2021-02-01
Series:Clinical and Translational Medicine
Online Access:https://doi.org/10.1002/ctm2.301
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spelling doaj-e0968a20b3ae4331b3998b83cf5746ef2021-02-26T10:40:39ZengWileyClinical and Translational Medicine2001-13262021-02-01112n/an/a10.1002/ctm2.301Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cellsShijun Wang0Leilei Ma1Ji'e Yang2Zhen Dong3Jian Wu4Xiancheng Lu5Jiahong Wang6Fangjie Dai7Guowei Tu8Lei Xu9Gang Zhao10Feng Zhang11Yunzeng Zou12Junbo Ge13Shanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaDepartment of outpatient Shanghai Yangpu Hospital of Traditional Chinese Medicine Shanghai ChinaDepartment of Cardiology Yangpu Hospital, Tongji University School of Medicine Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai ChinaShanghai Institute of Cardiovascular Diseases, Zhongshan Hospital Fudan University Shanghai Chinahttps://doi.org/10.1002/ctm2.301
collection DOAJ
language English
format Article
sources DOAJ
author Shijun Wang
Leilei Ma
Ji'e Yang
Zhen Dong
Jian Wu
Xiancheng Lu
Jiahong Wang
Fangjie Dai
Guowei Tu
Lei Xu
Gang Zhao
Feng Zhang
Yunzeng Zou
Junbo Ge
spellingShingle Shijun Wang
Leilei Ma
Ji'e Yang
Zhen Dong
Jian Wu
Xiancheng Lu
Jiahong Wang
Fangjie Dai
Guowei Tu
Lei Xu
Gang Zhao
Feng Zhang
Yunzeng Zou
Junbo Ge
Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
Clinical and Translational Medicine
author_facet Shijun Wang
Leilei Ma
Ji'e Yang
Zhen Dong
Jian Wu
Xiancheng Lu
Jiahong Wang
Fangjie Dai
Guowei Tu
Lei Xu
Gang Zhao
Feng Zhang
Yunzeng Zou
Junbo Ge
author_sort Shijun Wang
title Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
title_short Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
title_full Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
title_fullStr Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
title_full_unstemmed Activation of CXCL16/CXCR6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the IL‐17a‐producing CD1d+ T cells
title_sort activation of cxcl16/cxcr6 axis aggravates cardiac ischemia/reperfusion injury by recruiting the il‐17a‐producing cd1d+ t cells
publisher Wiley
series Clinical and Translational Medicine
issn 2001-1326
publishDate 2021-02-01
url https://doi.org/10.1002/ctm2.301
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