Shigella dysenteriae infection activates proinflammatory response through β-catenin/NF-κB signaling pathway.
Shigella dysenteriae (S.dysenteriae) the causative agent of bacillary dysentery invades the human colonic epithelium resulting in severe intestinal inflammatory response and epithelial destruction. However, the mechanism by which S.dysenteriae infection regulates proinflammatory cytokines during int...
Main Authors: | Ashidha Gopal, Iyer Soumya Chidambaram, Niranjali Devaraj, Halagowder Devaraj |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2017-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC5400225?pdf=render |
Similar Items
-
Shigella dysenteriae modulates BMP pathway to induce mucin gene expression in vivo and in vitro.
by: Ashidha Gopal, et al.
Published: (2014-01-01) -
Up-regulation of MUC2 and IL-1β expression in human colonic epithelial cells by Shigella and its interaction with mucins.
by: Radhakrishnan Prakash, et al.
Published: (2011-01-01) -
Iron acquisition by Shigella dysenteriae and Shigella flexneri
by: Davies, Nicola Mary Lisa
Published: (2008) -
A characterization of the lipids of Shigella dysenteriae /
by: Mann, Jerry Marvin
Published: (1967) -
Quercetin Suppresses Twist to Induce Apoptosis in MCF-7 Breast Cancer Cells.
by: Santhalakshmi Ranganathan, et al.
Published: (2015-01-01)