Amygdalin promotes the activity of T cells to suppress the progression of HBV-related hepatocellular carcinoma via the JAK2/STAT3 signaling pathway

Amygdalin promotes the activity of T cells to suppress the progression of HBV-related hepatocellular carcinoma via the JAK2/STAT3 signaling pathway

Abstract Background Hepatitis B virus (HBV) infection is a high-risk factor of hepatocellular carcinoma (HCC). Cellular immune responses are essential for HCC development, and the CD4+ and CD8+ T subtypes are identified as the primary anti-tumor immune cells. In the study, we investigated the effect...

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Bibliographic Details
Main Authors: Ruoyu Wang, Dong Zhang, Kewei Sun, Jianping Peng, Wenfang Zhu, Sihan Yin, Dan Tang, Yunan Wu
Format: Article
Language:English
Published: BMC 2021-01-01
Series:BMC Infectious Diseases
Subjects:
Hepatitis B virus (HBV)
Hepatocellular carcinoma (HCC)
Amygdalin
T cell
The JAK2/STAT3 signaling
Online Access:https://doi.org/10.1186/s12879-020-05713-0

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