Sestrin2 protects dendrite cells against ferroptosis induced by sepsis
Abstract Ferroptosis is a nonapoptotic form of programmed cell death triggered by the accumulation of reactive oxygen species (ROS) depended on iron overload. Although most investigations focus on the relationship between ferroptosis and cancer, neurodegenerative diseases, and ischemia/reperfusion i...
Main Authors: | Jing-yan Li, Chao Ren, Li-Xue Wang, Ren-qi Yao, Ning Dong, Yao Wu, Ying-ping Tian, Yong-ming Yao |
---|---|
Format: | Article |
Language: | English |
Published: |
Nature Publishing Group
2021-09-01
|
Series: | Cell Death and Disease |
Online Access: | https://doi.org/10.1038/s41419-021-04122-8 |
Similar Items
-
Antagonism of Cerebral High Mobility Group Box 1 Ameliorates Dendritic Cell Dysfunction in Sepsis
by: Chao Ren, et al.
Published: (2021-08-01) -
Sestrin2: Its Potential Role and Regulatory Mechanism in Host Immune Response in Diseases
by: Li-Xue Wang, et al.
Published: (2019-12-01) -
Ferroptosis: A Trigger of Proinflammatory State Progression to Immunogenicity in Necroinflammatory Disease
by: Jing-yan Li, et al.
Published: (2021-08-01) -
Comparison of clinical laboratory tests between bacterial sepsis and SARS-CoV-2-associated viral sepsis
by: Chao Ren, et al.
Published: (2020-08-01) -
Bilirubin Protects Transplanted Islets by Targeting Ferroptosis
by: Qing Yao, et al.
Published: (2020-06-01)