HDAC6 regulates LPS-tolerance in astrocytes.
Inflammatory tolerance is a crucial mechanism that limits inflammatory responses in order to avoid prolonged inflammation that may damage the host. Evidence that chronic inflammation contributes to the neuropathology of prevalent neurodegenerative and psychiatric diseases suggests that inflammatory...
Main Author: | Eléonore Beurel |
---|---|
Format: | Article |
Language: | English |
Published: |
Public Library of Science (PLoS)
2011-01-01
|
Series: | PLoS ONE |
Online Access: | http://europepmc.org/articles/PMC3192131?pdf=render |
Similar Items
-
HDAC3 Mediates the Inflammatory Response and LPS Tolerance in Human Monocytes and Macrophages
by: Mohammed Ghiboub, et al.
Published: (2020-10-01) -
Regulation by glycogen synthase kinase-3 of inflammation and T cells in CNS diseases
by: Eleonore eBeurel
Published: (2011-08-01) -
Astrocytes modulate the polarization of CD4+ T cells to Th1 cells.
by: Eléonore Beurel, et al.
Published: (2014-01-01) -
Lipopolysaccharide-induced interleukin-6 production is controlled by glycogen synthase kinase-3 and STAT3 in the brain
by: Jope Richard S, et al.
Published: (2009-03-01) -
Class I HDAC inhibition is a novel pathway for regulating astrocytic apoE secretion.
by: Erica Dresselhaus, et al.
Published: (2018-01-01)