Bmal1 Regulates the Redox Rhythm of HSPB1, and Homooxidized HSPB1 Attenuates the Oxidative Stress Injury of Cardiomyocytes
Oxidative stress is the main cause of acute myocardial infarction (AMI), which is related to the disorder of the regulation of Bmal1 on the redox state. HSPB1 form homologous-oxidized HSPB1 (homooxidized HSPB1) to resist oxidative damage via S-thiolated modification. However, it is still unclarified...
Main Authors: | Xiehong Liu, Wen Xiao, Yu Jiang, Lianhong Zou, Fang Chen, Weiwei Xiao, Xingwen Zhang, Yan Cao, Lei Xu, Yimin Zhu |
---|---|
Format: | Article |
Language: | English |
Published: |
Hindawi Limited
2021-01-01
|
Series: | Oxidative Medicine and Cellular Longevity |
Online Access: | http://dx.doi.org/10.1155/2021/5542815 |
Similar Items
-
HSPB1, HSPB6, HSPB7 and HSPB8 protect against RhoA GTPase-induced remodeling in tachypaced atrial myocytes.
by: Lei Ke, et al.
Published: (2011-01-01) -
HspB1, HspB5 and HspB4 in Human Cancers: Potent Oncogenic Role of Some of Their Client Proteins
by: André-Patrick Arrigo, et al.
Published: (2014-02-01) -
The Role of the Arginine in the Conserved N-Terminal Domain RLFDQxFG Motif of Human Small Heat Shock Proteins HspB1, HspB4, HspB5, HspB6, and HspB8
by: Vladislav M. Shatov, et al.
Published: (2018-07-01) -
HSPB1 facilitates the formation of non-centrosomal microtubules.
by: Leonardo Almeida-Souza, et al.
Published: (2013-01-01) -
Characterization of Hspb8 in Zebrafish
by: Magda Dubińska-Magiera, et al.
Published: (2020-06-01)