Dystrophin is required for the normal function of the cardio-protective K(ATP) channel in cardiomyocytes.

Dystrophin is required for the normal function of the cardio-protective K(ATP) channel in cardiomyocytes.

Duchenne and Becker muscular dystrophy patients often develop a cardiomyopathy for which the pathogenesis is still unknown. We have employed the murine animal model of Duchenne muscular dystrophy (mdx), which develops a cardiomyopathy that includes some characteristics of the human disease, to study...

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Bibliographic Details
Main Authors: Laura Graciotti, Jodi Becker, Anna Luisa Granata, Antonio Domenico Procopio, Lino Tessarollo, Gianluca Fulgenzi
Format: Article
Language:English
Published: Public Library of Science (PLoS) 2011-01-01
Series:PLoS ONE
Online Access:http://europepmc.org/articles/PMC3205025?pdf=render

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