Staphylococcus aureus Exploits the Host Apoptotic Pathway To Persist during Infection
Caspase-3 controls the apoptotic pathway, a form of programmed cell death designed to be immunologically silent. Polymorphisms leading to reduced caspase-3 activity are associated with variable effects on tumorigenesis and yet arise frequently. Staphylococcus aureus is a human commensal and a freque...
Main Authors: | Volker Winstel, Olaf Schneewind, Dominique Missiakas |
---|---|
Format: | Article |
Language: | English |
Published: |
American Society for Microbiology
2019-11-01
|
Series: | mBio |
Subjects: | |
Online Access: | https://doi.org/10.1128/mBio.02270-19 |
Similar Items
-
Selective Host Cell Death by Staphylococcus aureus: A Strategy for Bacterial Persistence
by: Dominique Missiakas, et al.
Published: (2021-01-01) -
Staphylococcus aureus induces neutrophil extracellular traps (NETs) and neutralizes their bactericidal potential
by: Pietro Speziale, et al.
Published: (2021-01-01) -
Neutrophil dynamics in the blood and milk of crossbred cows naturally infected with Staphylococcus aureus
by: Dilip K. Swain, et al.
Published: (2015-03-01) -
Adenosine synthase A contributes to recurrent Staphylococcus aureus infection by dampening protective immunity
by: Jian Deng, et al.
Published: (2021-08-01) -
Septal secretion of protein A in Staphylococcus aureus requires SecA and lipoteichoic acid synthesis
by: Wenqi Yu, et al.
Published: (2018-05-01)