Activation of γ-aminobutyric Acid (A) Receptor Protects Hippocampus from Intense Exercise-induced Synapses Damage and Apoptosis in Rats

Activation of γ-aminobutyric Acid (A) Receptor Protects Hippocampus from Intense Exercise-induced Synapses Damage and Apoptosis in Rats

Background: Our previous study has confirmed that one bout of exhaustion (Ex) can cause hippocampus neurocyte damage, excessive apoptosis, and dysfunction. Its initial reason is intracellular calcium overload in hippocampus triggered by N-methyl-D-aspartic acid receptor (NMDAR) over-activation. NMDA...

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Bibliographic Details
Main Authors: Yi Ding, Lan Xie, Cun-Qing Chang, Zhi-Min Chen, Hua Ai
Format: Article
Language:English
Published: Wolters Kluwer 2015-01-01
Series:Chinese Medical Journal
Subjects:
Endoplasmic Reticulum Stress-induced Apoptosis; γ-aminobutyric Acid (A) Receptor; Hippocampus; Intense Exercise; Synapse Plasticity
Online Access:http://www.cmj.org/article.asp?issn=0366-6999;year=2015;volume=128;issue=17;spage=2330;epage=2339;aulast=Ding

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