<i>Helicobacter pylori</i> Colonization Drives Urokinase Receptor (uPAR) Expression in Murine Gastric Epithelium During Early Pathogenesis

<i>Helicobacter pylori</i> Colonization Drives Urokinase Receptor (uPAR) Expression in Murine Gastric Epithelium During Early Pathogenesis

(1) Background: Persistent <i>Helicobacter pylori</i> infection is the most important risk factor for gastric cancer. The urokinase receptor (uPAR) is upregulated in lesions harboring cancer invasion and inflammation. Circumstantial evidence tends to correlate <i>H. pylori</i>...

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Bibliographic Details
Main Authors: Warner Alpízar-Alpízar, Mette E. Skindersoe, Lone Rasmussen, Mette C. Kriegbaum, Ib J. Christensen, Ida K. Lund, Martin Illemann, Ole D. Laerum, Karen A. Krogfelt, Leif P. Andersen, Michael Ploug
Format: Article
Language:English
Published: MDPI AG 2020-07-01
Series:Microorganisms
Subjects:
uPAR
<i>Helicobacter pylori</i>
gastric cancer
gastritis
mucous metaplasia
mouse model
Online Access:https://www.mdpi.com/2076-2607/8/7/1019

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