Behavioral neuroadaptation to alcohol : from glucocorticoids to histone acetylation
A prime mechanism that contributes to the development and maintenance of alcoholism is the dysregulation of the hypothalamic-pituitary-adrenal (HPA) axis activity and the release of glucocorticoids (cortisol in humans and primates, corticosterone in rodents) from the adrenal glands. In the brain, su...
Main Authors: | Daniel Beracochea, Nicole Mons |
---|---|
Format: | Article |
Language: | English |
Published: |
Frontiers Media S.A.
2016-10-01
|
Series: | Frontiers in Psychiatry |
Subjects: | |
Online Access: | http://journal.frontiersin.org/Journal/10.3389/fpsyt.2016.00165/full |
Similar Items
-
Targeting the Glucocorticoid Receptors During Alcohol Withdrawal to Reduce Protracted Neurocognitive Disorders
by: Daniel Béracochéa, et al.
Published: (2019-09-01) -
Role of non-coding RNAs in the Neuroadaptation to Alcoholism and Fetal Alcohol Exposure
by: Matthew eReilly
Published: (2012-06-01) -
Non-canonical Glucocorticoid Receptor Transactivation of gilz by Alcohol Suppresses Cell Inflammatory Response
by: Hang Pong Ng, et al.
Published: (2017-06-01) -
Transcriptional Correlates of Chronic Alcohol Neuroadaptation in Drosophila Larvae
by: Acevedo-Gonzalez, J.P, et al.
Published: (2021) -
Potential role of adolescent alcohol exposure-induced amygdaloid histone modifications in anxiety and alcohol intake during adulthood
by: Subhash C. Pandey, et al.
Published: (2015-10-01)