Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore

Bibliographic Details
Main Authors: Elena C. Castillo, José A. Morales, Héctor Chapoy-Villanueva, Christian Silva-Platas, Niria Treviño-Saldaña, C. Enrique Guerrero-Beltrán, Judith Bernal-Ramírez, Alejandro Torres-Quintanilla, Noemí García, Keith Youker, Guillermo Torre-Amione, Gerardo García-Rivas
Format: Article
Language:English
Published: Cell Physiol Biochem Press GmbH & Co KG 2019-08-01
Series:Cellular Physiology and Biochemistry
Online Access:https://www.cellphysiolbiochem.com/Articles/000151/
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spelling doaj-6b320f23ab5349b8a15e3de955186f512020-11-25T03:08:49ZengCell Physiol Biochem Press GmbH & Co KGCellular Physiology and Biochemistry1015-89872019-08-0153346547910.33594/000000151Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition PoreElena C. Castillo0José A. Morales1Héctor Chapoy-Villanueva2Christian Silva-Platas3Niria Treviño-Saldaña4C. Enrique Guerrero-Beltrán5Judith Bernal-Ramírez6Alejandro Torres-Quintanilla7Noemí García8Keith Youker9Guillermo Torre-Amione10Gerardo García-Rivas11Tecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, México; Tecnologico de Monterrey, Hospital Zambrano Hellion, TecSalud, Centro de Investigación Biomédica, San Pedro Garza García, México Tecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, MéxicoTecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, México; Tecnologico de Monterrey, Hospital Zambrano Hellion, TecSalud, Centro de Investigación Biomédica, San Pedro Garza García, MéxicoWeill Cornell Medical College, Methodist DeBakey Heart & Vascular Center, The Methodist Hospital, Houston, TX, USATecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, México; Tecnologico de Monterrey, Hospital Zambrano Hellion, TecSalud, Centro de Investigación Biomédica, San Pedro Garza García, México; Weill Cornell Medical College, Methodist DeBakey Heart & Vascular Center, The Methodist Hospital, Houston, TX, USATecnologico de Monterrey, Escuela de Medicina y Ciencias de la Salud, Medicina Cardiovascular y Metabolómica, Monterrey, México; Tecnologico de Monterrey, Hospital Zambrano Hellion, TecSalud, Centro de Investigación Biomédica, San Pedro Garza García, Méxicohttps://www.cellphysiolbiochem.com/Articles/000151/
collection DOAJ
language English
format Article
sources DOAJ
author Elena C. Castillo
José A. Morales
Héctor Chapoy-Villanueva
Christian Silva-Platas
Niria Treviño-Saldaña
C. Enrique Guerrero-Beltrán
Judith Bernal-Ramírez
Alejandro Torres-Quintanilla
Noemí García
Keith Youker
Guillermo Torre-Amione
Gerardo García-Rivas
spellingShingle Elena C. Castillo
José A. Morales
Héctor Chapoy-Villanueva
Christian Silva-Platas
Niria Treviño-Saldaña
C. Enrique Guerrero-Beltrán
Judith Bernal-Ramírez
Alejandro Torres-Quintanilla
Noemí García
Keith Youker
Guillermo Torre-Amione
Gerardo García-Rivas
Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
Cellular Physiology and Biochemistry
author_facet Elena C. Castillo
José A. Morales
Héctor Chapoy-Villanueva
Christian Silva-Platas
Niria Treviño-Saldaña
C. Enrique Guerrero-Beltrán
Judith Bernal-Ramírez
Alejandro Torres-Quintanilla
Noemí García
Keith Youker
Guillermo Torre-Amione
Gerardo García-Rivas
author_sort Elena C. Castillo
title Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
title_short Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
title_full Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
title_fullStr Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
title_full_unstemmed Mitochondrial Hyperacetylation in the Failing Hearts of Obese Patients Mediated Partly by a Reduction in SIRT3: The Involvement of the Mitochondrial Permeability Transition Pore
title_sort mitochondrial hyperacetylation in the failing hearts of obese patients mediated partly by a reduction in sirt3: the involvement of the mitochondrial permeability transition pore
publisher Cell Physiol Biochem Press GmbH & Co KG
series Cellular Physiology and Biochemistry
issn 1015-8987
publishDate 2019-08-01
url https://www.cellphysiolbiochem.com/Articles/000151/
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