Acute liver injury following acetaminophen administration does not activate atrophic pathways in the mouse diaphragm
Abstract N-acetyl-para-amino phenol (APAP, usually named paracetamol), which is commonly used for its analgesic and antipyretic properties may lead to hepatotoxicity and acute liver damage in case of overdoses. Released cytokines and oxidative stress following acute liver damage may affect other org...
Main Authors: | C. S. Bruells, P. Duschner, G. Marx, G. Gayan-Ramirez, N. Frank, T. Breuer, O. Krenkel, F. Tacke, J. C. Mossanen |
---|---|
Format: | Article |
Language: | English |
Published: |
Nature Publishing Group
2021-03-01
|
Series: | Scientific Reports |
Online Access: | https://doi.org/10.1038/s41598-021-85859-2 |
Similar Items
-
Adapted Immune Responses of Myeloid-Derived Cells in Fatty Liver Disease
by: Jana Hundertmark, et al.
Published: (2018-10-01) -
Acetaminophen for analgesia following pyloromyotomy: does the route of administration make a difference?
by: Yung A, et al.
Published: (2016-03-01) -
Protein turnover in the isolated mouse diaphragm
by: Mulligan, Mark Thomas
Published: (1987) -
Diaphragm Muscle Fatigue Resistance Does Not Increase With Age
by: Matthew J Fogarty, et al.
Published: (2020-01-01) -
Protective effect of pinocembrin in a mouse model of liver injury induced by acetaminophen
by: DU Yichao
Published: (2020-03-01)