Rapid <it>S</it>-nitrosylation of actin by NO-generating donors and in inflammatory pain model mice

Rapid <it>S</it>-nitrosylation of actin by NO-generating donors and in inflammatory pain model mice

<p>Abstract</p> <p>Background</p> <p><it>S</it>-Nitrosylation, the reversible post-translational modification of reactive cysteine residues in proteins, has emerged as an important mechanism by which NO acts as a signaling molecule. We recently demonstrated...

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Bibliographic Details
Main Authors: Lu Jingshan, Katano Tayo, Uta Daisuke, Furue Hidemasa, Ito Seiji
Format: Article
Language:English
Published: SAGE Publishing 2011-12-01
Series:Molecular Pain
Subjects:
dopamine release
F-actin
inflammatory pain
nitric oxide
PC12 cell
<it>S</it>-nitrosylation
spinal cord
<it>in vivo </it>patch-clamp recordings
Online Access:http://www.molecularpain.com/content/7/1/101

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http://www.molecularpain.com/content/7/1/101

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