Increased susceptibility of spinal muscular atrophy fibroblasts to camptothecin is p53-independent
<p>Abstract</p> <p>Background</p> <p>Deletion or mutation(s) of the survival motor neuron 1 <it>(SMN1) </it>gene causes spinal muscular atrophy (SMA). The SMN protein is known to play a role in RNA metabolism, neurite outgrowth, and cell survival. Yet, it re...
| Main Authors: | , , , , |
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| Format: | Article |
| Language: | English |
| Published: |
BMC
2009-05-01
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| Series: | BMC Cell Biology |
| Online Access: | http://www.biomedcentral.com/1471-2121/10/40 |